Muscular Disorders, Atrophic
"Muscular Disorders, Atrophic" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Disorders characterized by an abnormal reduction in muscle volume due to a decrease in the size or number of muscle fibers. Atrophy may result from diseases intrinsic to muscle tissue (e.g., MUSCULAR DYSTROPHY) or secondary to PERIPHERAL NERVOUS SYSTEM DISEASES that impair innervation to muscle tissue (e.g., MUSCULAR ATROPHY, SPINAL).
| Descriptor ID |
D020966
|
| MeSH Number(s) |
C05.651.534 C10.668.491.175
|
| Concept/Terms |
Muscular Disorders, Atrophic- Muscular Disorders, Atrophic
- Atrophic Muscular Disorder
- Disorder, Atrophic Muscular
- Disorders, Atrophic Muscular
- Muscular Disorder, Atrophic
- Atrophic Muscular Disorders
Spinopontine Atrophy- Spinopontine Atrophy
- Atrophies, Spinopontine
- Spinopontine Atrophies
- Atrophy, Spinopontine
Atrophy, Muscular, Spinobulbar- Atrophy, Muscular, Spinobulbar
- Spinobulbar Muscular Atrophy
- Spinobulbar Atrophy
- Atrophies, Spinobulbar
- Atrophy, Spinobulbar
- Spinobulbar Atrophies
- Muscular Atrophy, Spinobulbar
- Atrophies, Spinobulbar Muscular
- Atrophy, Spinobulbar Muscular
- Muscular Atrophies, Spinobulbar
- Spinobulbar Muscular Atrophies
- Spinal and Bulbar Muscular Atrophy
Atrophy, Disuse- Atrophy, Disuse
- Atrophies, Disuse
- Disuse Atrophies
- Disuse Atrophy
|
Below are MeSH descriptors whose meaning is more general than "Muscular Disorders, Atrophic".
Below are MeSH descriptors whose meaning is more specific than "Muscular Disorders, Atrophic".
This graph shows the total number of publications written about "Muscular Disorders, Atrophic" by people in this website by year, and whether "Muscular Disorders, Atrophic" was a major or minor topic of these publications.
View timeline visualization
| Year | Major Topic | Minor Topic | Total |
|---|
| 2003 | 2 | 1 | 3 |
| 2007 | 1 | 0 | 1 |
| 2012 | 0 | 1 | 1 |
| 2013 | 1 | 0 | 1 |
| 2014 | 3 | 0 | 3 |
| 2015 | 1 | 0 | 1 |
| 2016 | 1 | 0 | 1 |
| 2017 | 1 | 0 | 1 |
| 2018 | 2 | 0 | 2 |
| 2019 | 1 | 0 | 1 |
| 2023 | 1 | 0 | 1 |
| 2024 | 1 | 0 | 1 |
Below are the most recent publications written about "Muscular Disorders, Atrophic" by people in Profiles.
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Transcriptome and Metabolome Profiling Provide New Insights into Disuse Muscle Atrophy in Chicken: The Potential Role of Fast-Twitch Muscle Fibers. Int J Mol Sci. 2024 Mar 20; 25(6).
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Skeletal muscle-specific DJ-1 ablation-induced atrogenes expression and mitochondrial dysfunction contributing to muscular atrophy. J Cachexia Sarcopenia Muscle. 2023 10; 14(5):2126-2142.
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Non-fibro-adipogenic pericytes from human embryonic stem cells attenuate degeneration of the chronically injured mouse muscle. JCI Insight. 2019 12 19; 4(24).
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Diffusion tensor imaging and diffusion modeling: Application to monitoring changes in the medial gastrocnemius in disuse atrophy induced by unilateral limb suspension. J Magn Reson Imaging. 2019 06; 49(6):1655-1664.
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A mini review: Proteomics approaches to understand disused vs. exercised human skeletal muscle. Physiol Genomics. 2018 09 01; 50(9):746-757.
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Relationship of changes in strain rate indices estimated from velocity-encoded MR imaging to loss of muscle force following disuse atrophy. Magn Reson Med. 2018 02; 79(2):912-922.
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Adenylyl cyclase activating polypeptide reduces phosphorylation and toxicity of the polyglutamine-expanded androgen receptor in spinobulbar muscular atrophy. Sci Transl Med. 2016 12 21; 8(370):370ra181.
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Control of skeletal muscle atrophy in response to disuse: clinical/preclinical contentions and fallacies of evidence. Am J Physiol Endocrinol Metab. 2016 09 01; 311(3):E594-604.
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Glycolytic-to-oxidative fiber-type switch and mTOR signaling activation are early-onset features of SBMA muscle modified by high-fat diet. Acta Neuropathol. 2016 07; 132(1):127-44.
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Nemo-like kinase is a novel regulator of spinal and bulbar muscular atrophy. Elife. 2015 Aug 26; 4:e08493.