Tangcay Tenorio. Teves Group 4 C1 Concept Map On Hydatidiform Mole
Tangcay Tenorio. Teves Group 4 C1 Concept Map On Hydatidiform Mole
COLLEGE OF NURSING
Silliman University
Dumaguete City
CONCEPT MAP
ON
HYDATIDIFORM MOLE
NCM 34 C1- LEVEL II
Submitted by:
Hydatidiform Mole
Hydatidiform moles are type of gestational trophoblastic disease. The condition is also known
as molar pregnancy. It is an abnormal fertilized egg that develops into a hydatidiform mole
instead of fetus. Hydatidiform mole can develop from cells called trophoblast (forms the
placenta and amniotic sac) that remain in the uterus after a miscarriage, full term pregnancy or
ectopic pregnancy. The affected cells grow abnormally and multiply quickly. Hydatidiform
mole rarely develops when there is a living fetus. In some cases, the fetus dies and miscarriage
occurs. It is common in women under 17 and over 35. Hydatidiform moles are almost 10 times
common in Asian countries. About 2-3% of hydatidiform moles develop into choriocarcinoma.
Symptoms of hydatidiform mole include n/v and vaginal bleeding. Complications include
infection, sepsis, shock, hyperthyroidism, tachycardia, preeclampsia and choriocarcinoma.
Hydatidiform mole is completely removed usually by Dilatation and curettage with suction.
Removal of uterus (hysterectomy) is rarely done. Chemotherapy is needed if the mole persists.
Chemotherapy may consist of only one drug (methotrexate or dactinomycin).
The hydatidiform mole (HM) is a placental pathology of androgenetic origin. Placental villi
have an abnormal hyperproliferation event and hydropic degeneration. Three situations can be
envisaged at its origin: 1. The destruction/expulsion of the female pronucleus at the time of
fertilization by 1 or 2 spermatozoa with the former being followed by an endoreplication of
the male pronucleus leading to a complete hydatidiform mole (CHM) 2. A triploid zygote
(fertilization by 2 spermatozoa) leading to a partial hydatidiform mole (PHM) but can also lead
to haploid and diploid clones. The diploid clone may produce a normal fetus while the haploid
clone after endoreplication generates a CHM 3. A nutritional defect during the differentiation
of the oocytes or the deterioration of the limited oxygen pressure during the first trimester of
gestation may lead to the formation of a HM. Numerous risk factors for molar pregnancies
have been suggested, including paternal age, maternal genetic anomalies, blood group, oral
contraceptives, maternal age and environmental factors; in particular vitamin A and the folates.
A deficit of vitamin A or/and of folates during the period of 18 to 21 d of gestation is associated
with an absence of vascularization of the placental villosities, which is observed in the CHM.
It is also noted that a reduction in vitamin A in the food of the patients at the time of their
pregnancy could explain the geographical distribution of these moles.
Hydatidiform Mole
- It is a growth of an abnormal fertilized egg or an overgrowth of tissue from
the placenta.
Medical Management
1. Induced abortion
2. Follow-up care because of the risk for developing choriocarcinoma
3. Weekly monitoring of hCG until they remain normal for 3 consecutive weeks
4. Periodic follow-up for 1 up to 3 years
5. Avoidance of pregnancy until HCG levels are normal
6. Evacuation of the uterus by dilation and curettage.
7. Prophylactic total hysterectomy
8. Prophylactic chemotherapy after evacuation of hydatidiform mole.
9. Consideration of using other uterotonic formulations (eg, Methergine, Hemabate).
Nursing Management
Pillitteri, A., & Silbert-Flagg, J. (2018). Maternal and Child Health Nursing: Care of the
Childbearing and Childrearing Family. 8th ed. USA. Wolters Kluwer
Ramirez, P., & Salvo, G. (n.d.). Hydatidiform mole - women's health issues. Retrieved February
17, 2021, from https://www.msdmanuals.com/home/women-s-health-issues/cancers-of-the-
female-reproductive-system/hydatidiform-
mole#:~:text=A%20hydatidiform%20mole%20is%20growth,and%20very%20high%20blo
od%20pressure
The Hydatidiform Mole (The hydatidiform mole. (2016). Cell Adhesion & Migration. Retrieved from
https://www.tandfonline.com/doi/full/10.1080/19336918.2015.1093275)
HYDATIDIFORM MOLE (CASE SCENARIO)
Anne, a 17-year-old nulliparous adolescent girl presented with progressive lower abdominal pain and pronounced nausea and vomiting of about a
week’s duration. Her gestational age was 12 weeks and 4 days according to her last menstruation date. Pelvic examination revealed a closed cervical
os and no uterine bleeding. Subsequent ultrasound and magnetic resonance imaging (MRI) depicted a T2- hyperintense, huge intrauterine mass,
numerous cystic spaces, and massive peri-lesion hemorrhage. Immediately after introducing forceps into the uterine cavity for suction curettage, a large
amount of trophoblastic tissue was expelled and sudden gushes of blood with clots were passed. Histological examination resulted in a final diagnosis
of complete hydatidiform mole.
CUES|EVIDENCES NURSING GOALS NURSING RATIONALE EVALUATION
DIAGNOSIS INTERVENTIONS
● Early
● Observe for identification as
bleeding a result of
tendencies cancer. Allows
prompt
intervention.
(TANGCAY) Acute pain r/t Within my 8 hours of ● Administer ● To alleviate At the end of my 8-
Objective: hydatidiform mole as care, patient will prescribed acute pelvic hour care, the goals
● Lower evidence by lower demonstrate relief of pain medications as pain are met as evidenced
abdominal pain abdominal pain and as evidenced by: per Doctor’s by:
● MRI results verbalization of order PRN ● A pain score
(huge pelvic pain ● A pain score of 0 ● Assess ● For baseline of 0 out of 10
intrauterine out of 10 patient’s vital data. The tome ● Stable vital
mass and ● Stable vital signs signs at least of monitoring of signs
massive peri- ● Absence of 30 minutes vital signs may ● Absence of
lesion restlessness after depend on the restlessness
hemorrhage) ● Verbalizations of administration peak time of ● Verbalizations
comfort of medication drug of comfort
Subjective: administered ● Ability to sleep
“Sakit na jud kaayo ● Assess for ● To monitor and rest
akong pus-on nars, pain (e.g. pain effectiveness of
gusto ko mulimbag sa scale) at least medical
kasakit.” 30 minutes treatment for
after the relief of
medication pelvic pain.
intake
● Elevate the ● To increase the
head the oxygen level by
patient in allowing optimal
semi-Fowler’s lung expansion
● To provide
● Place the optimal comfort
patient in to the patient
complete bed and avoid risk
rest during for injury
severe
episodes of
pain ● Deep breathing
● Perform non- exercises,
pharmacologic guided imagery
al relief and provision of
measures distractions
such as such as TV or
relaxation radio
techniques ● To reduce post-
● After surgery, surgical pain
advise the and allow full
patient to have recovery and
no strenuous healing
activity for a
few weeks