Walter M Chesnut: @parsifaler

Walter M Chesnut
@Parsifaler
Apr 4 • 11 tweets •
Parsifaler/status/1510789756690571270
1) PLEASE READ THIS ENTIRE THREAD. IT IS

URGENT.
Building on all recent work. By far my most important

synthesis:
A NEW KIND OF CANCER: THE MARRIAGE OF

ONCOGENESIS AND PRIONOPATHY
THE PROGRESSIVE DESTRUCTION OF TISSUE AND

ORGANS BY THE AMYLOIDOGENIC AND
OLIGOMERIC PROPERTIES OF
2) THE SARS-CoV-2 SPIKE PROTEIN
It has been proposed that Alzheimer's disease might be a 'whole body' problem, as
amyloid-beta can travel, cancer-like, to brain from other parts of body.
Normal mice that had been joined (via bloodstreams) to genetically modified
partners for a
3) year "contracted" Alzheimer's disease. The researcher song says the amyloid-beta
traveled from the genetically-modified mice to the brains of their normal partners,
where it accumulated and began to inflict damage.
Not only did the normal mice develop plaques, but also a
4) pathology similar to "TANGLES" -- twisted protein strands that form inside brain
cells (please note, per image, this is PRECISELY what the Spike Protein induces),
disrupting their function and eventually killing them from the inside-out. Other signs
of Alzheimer's-like damage
5) included brain cell degeneration, inflammation and microbleeds. In addition, the

ability to transmit electrical signals involved in learning and memory -- a sign of a
healthy brain -- was impaired, even in mice that had been joined for just four months.
Besides the brain,
6) amyloid-beta is produced in blood platelets, blood vessels and muscles, and its
precursor protein is found in several other organs. But until these experiments, it was
unclear if amyloid-beta from outside the brain could contribute to Alzheimer's
disease.
Which brings us to
7) the Fibrils that the Spike Protein is inducing.
Studies of nerve biopsy or cardiac autopsy specimens from patients with ATTR and
AL amyloidoses show atrophy of cells near amyloid fibril aggregates. In addition to
the stress or toxicity attributable to amyloid fibrils
8) themselves, the toxicity of non-fibrillar states of amyloidogenic proteins,

PARTICULARLY OLIGOMERS, may also participate in the mechanisms of tissue
damage.
By the way. Who discovered this? Department of Neurology and Centre for Clinical
Neuroscience, Daping Hospital, THIRD
9) MILITARY MEDICAL UNIVERSITY, Chongqing, China.
The proposed therapeutics to deal with this pathology sound quite familiar: (1)
reducing or preventing the production of causative proteins; (2) preventing the
causative proteins from participating in the process of amyloid fibril
10) formation; and/or (3) eliminating already-deposited amyloid fibrils.
And this is what is MOST ALARMING and should cause the world to go FULL
IMMEDIATE STOP ON ALL SPIKE PROTEIN THERAPIES. PLEASE READ (1)
AGAIN!
(1)REDUCING OR PREVENTING THE PRODUCTION OF CAUSITIVE PROTEINS.
11) We need to make ourselves well, before we make ourselves ever more ill.
The Ultrastructure of Tissue Damage by Amyloid Fibrils

Amyloidosis is a group of diseases that includes Alzheimer’s disease, prion
diseases, transthyretin (ATTR) amyloidosis, and immunoglobulin light chain (AL)
amyloidosis. The mechanism of organ dysfunc…
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8347239/
Alzheimer's disease might be a 'whole body' problem: Amyloid-beta ca…

Canadian and Chinese scientists, using surgically-joined mice, find that amyloid-
beta -- the protein that causes Alzheimer's disease -- can travel from other parts of
the body to the brain, where it …
https://www.sciencedaily.com/releases/2017/10/171031084808.htm
Blood-derived amyloid-β protein induces Alzheimer’s disease patholo…

The amyloid-β protein (Aβ) protein plays a pivotal role in the pathogenesis of
Alzheimer’s disease (AD). It is believed that Aβ deposited in the brain originates
from the brain tissue itself. However…
https://www.nature.com/articles/mp2017204
Amyloidogenesis of SARS-CoV-2 Spike Protein

SARS-CoV-2 infection is associated with a surprising number of morbidities.
Uncanny similarities with amyloid-disease associated blood coagulation and
fibrinolytic disturbances together with neurolog…
https://www.biorxiv.org/content/10.1101/2021.12.16.472920v1.full
Oligomerization of the SARS-CoV S glycoprotein: dimerization of the N…
Viral envelope glycoproteins are oligomeric and the quaternary structure is critical
for their membrane fusion activity. Typically the transmembrane glycoproteins of
class I fusion proteins contain t…
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7092807/
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Walter M Chesnut: @parsifaler

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Walter M Chesnut

1) PLEASE READ THIS ENTIRE THREAD. IT IS

Building on all recent work. By far my most important

A NEW KIND OF CANCER: THE MARRIAGE OF

THE PROGRESSIVE DESTRUCTION OF TISSUE AND

2) THE SARS-CoV-2 SPIKE PROTEIN

5) included brain cell degeneration, inflammation and microbleeds. In addition, the

Besides the brain,

7) the Fibrils that the Spike Protein is inducing.

8) themselves, the toxicity of non-fibrillar states of amyloidogenic proteins,

9) MILITARY MEDICAL UNIVERSITY, Chongqing, China.

10) formation; and/or (3) eliminating already-deposited amyloid fibrils.

(1)REDUCING OR PREVENTING THE PRODUCTION OF CAUSITIVE PROTEINS.

The Ultrastructure of Tissue Damage by Amyloid Fibrils

Alzheimer's disease might be a 'whole body' problem: Amyloid-beta ca…

Blood-derived amyloid-β protein induces Alzheimer’s disease patholo…

Amyloidogenesis of SARS-CoV-2 Spike Protein

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