Viterous Hemmorage
Viterous Hemmorage
Viterous Hemmorage
MADAN SN
53
VITREOUS HAEMORRHAGE
6. Exudative age-related macular degeneration usually high CNVM, may have vitreous
haemorrhage
7.Blood dyscrasias, e.g. retinopathy of anaemia, leukaemias, polycythemias and sickle-cell
retinopathy.
8. Bleeding disorders, e.g. purpura, haemophilia and scurvy.
9. Neoplasms. Vitreous haemorrhage may occur from rupture of vessels due to acute necrosis in
tumours like retinoblastoma and malignant melanoma of choroid.
10. Other causes include Coat's diseases, radiation retinopathy, retinal capillary aneurysm,
retinal macroaneurysm terson syndrome.
Clinical features
Depending upon the location it is labeled as:
• Anterior vitreous haemorrhage,
• Mid vitreous haemorrhage,
• Posterior vitreous haemorrhage, or
• Total vitreous haemorrhage
Symptoms
• Floaters of sudden onset occur when the vitreous haemorrhage is small
• Sudden painless loss of vision occurs in large vitreous haemorrhage.
Signs
• Distant direct ophthalmoscopy reveals black shadows against the red glow in small
haemorrhages and no red glow in a large haemorrhage.
• Direct and indirect ophthalmoscopy may show presence of blood in the vitreous cavity in
small vitreous haemorrhage and non-visualization of fundus in large vitreous haemorrhage.
• Slit-lamp examination shows normal anterior segment and a reddish mass in the vitreous.
• Ultrasonography with B-scan is particularly helpful in diagnosing vitreous haemorrhage.
Fate of vitreous haemorrhage
l. Complete absorption may occur without organization and the vitreous becomes clear within
4-8 weeks.
2. Organization of haemorrhage with formation of a yellowish-white debris occurs in persistent
or recurrent bleeding.
3. Complications like vitreous liquefaction, degeneration and khaki cell glaucoma (in aphakia)
may occur.
4. Retinitis proliferans may occur which may be complicated by tractional retinal detachment.
Treatment
I. Conservative treatment consists of bed rest and elevation of patient's head. This will allow
the blood to settle down. Most of the time conservative treatment is sufficient.
II. Treatment of the cause. Once the blood settles down, indirect ophthalmoscopy should be
performed to locate and further manage the causative lesion such as a retinal break,
phlebitis, proliferative retinopathy, etc.
III. Vitrectomy by pars plana route should be considered to clear the vitreous, if the
haemorrhage is not absorbed after 3 months. Early vitrectomy may be required when
associated with retinal detachment.
Reference
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