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CEREBRO VASCULAR ACCIDENT

INTRODUCTION

 Cerebro vascular accident is a sudden death of some brain


cells due to lack of oxygen when the blood flow to the
brain is impaired by blockage or rupture of an artery to
the brain.
 Cerebro vascular accident is also know as “ stroke” or “
brain attack”.
DEFINITION

According to WHO(1970)

Stroke or Cerebro vascular accident is rapidly developed


clinical signs focal or global disturbance of cerebral function
lasting more than 24 hours or leading to death with no
apparent cause other than of vascular origin.
ETIOLOGY AND RISK FACTORS

ETIOLOGY
Etiological factors can be classified into:

 ISCHEMIC STROKE
 HEMORRHAGIC STROKE
ISCHEMIC STROKE

About 80% of stroke are ischemic stroke. QIT occurs when


the arteries to your brain becomes narrowed or blocked
causing severely reduced blood flow. The most common
ischemic stroke include:
 THROMBOTIC STROKE- A thrombotic stroke occurs from
the injury to a blood vessel wall and formation of blood
clot.
 Thrombosis develops readily where atherosclerosis plaques
have already narrowed blood vessels.
• EMBOLIC STROKE

Embolic stroke occurs when an embolus lodges in and


occludes a cerebral artery, resulting in infraction and
edema of the area supplied by the involved vessel.
HEMORRHAGIC STROKE

Amounts for approx 15% of all strokes and results from


bleeding into the brain tissue itself or into the subarachnoid
space or ventricles. The most common type are:
 INTRACEREBRAL HEMORRHAGE: Is bleeding within the
brain caused by a rupture of a vessel.
 Hypertension is the most important cause of intracerebral
hemorrhage.
RISK FACTORS

Strokes risk increase with multiple risk factors:


 NON MODIFIABLE RISK FACTORS
1. Age
2. Gender
3. Race
4. Family history/ hereditary
 NON MODIFIABLE RISK FACTORS: Risks that can potentially be altered through
lifestyle and medical treatment which include-
1. Hypertension
2. Heart disease
3. Smoking
4. Excessive alcohol consumption
5. Obesity
6. Poor diet
7. Drug abuse
PATHOPHYSIOLOGY

1. Accumulation of lipids and inflammatory cells in intima of affected artery.


2. Carotid atherosclerosis.
3. Hypertrophy of arterial smooth muscle.
4. Plaque formation
5. Plaque rupture
6. Embolus
7. Arterial occlusion
8. Reduced blood flow
9. Ischemia
10. Cell necrosis.
CLINICAL MANIFESTATIONS
A stroke can have effect on many body functions including:
 MOTOR FUNCTIONS
1. Mobility
2. Respiratory functions
3. Swallowing and speech problems
4. Self care deficit

 COMMUNICATION
1. Aphasia (a language disorder that affects ability to communicate.
2. Dysphagia( Impairment in the production of speech).
 AFFECT: Emotional response may be exaggerated or
unpredictable.
 INTELLECTUAL FUNCTIONS: Both memory and judgement
maybe impaired.
 ELIMINATION: Most problems with urinary and bowel
elimination occur initially and are temporary.
DIAGNOSTIC EVALUATION

 Physical examination
 Blood tests
 CT scan
 MRI
 Carotid ultrasound
MANAGEMENT
 MEDICAL MANAGEMENT
1. Antiplatelet drug- Asprin 81-325 mg/day
2. Oral anticoagulant- Warfarin
 SURGICAL MANAGEMENT
1. Carotid endarterectomy (CEA)
 Transluminal angioplasty
 Stenting
 Extra cranial – intracranial bypass
NURSING MANAGEMENT
 Pain related to increased intracranial pressure as evidenced by
patient verbalisation.
 Hyperthermia related to disease condition as evidenced by raised
body temperature.
 Imbalanced nutrition less than body requirement related to less
intake of food as evidenced by weight loss .
 Disturbed sleep pattern related to hospitalisation as evidenced by
patient verbalisation.
COMPLICATION

 Paralysis or loss of muscle movement.


 Difficulty talking and swallowing
 Memory loss
 Emotional problems
 Behaviour changes.

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